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Description
Recent investigations have unearthed a novel player in the ferroptosis domain, MBOAT2, functioning autonomously of GPX4 and FSP1, overseeing ferroptosis surveillance through phospholipid remodeling and proficiently impeding ferroptotic processes
doi: 10.1016/j.cell.2016.06.056
USA 116 , 49464954 (2019)
While these future directions highlight the substantial promise of H therapy, several current limitations must still be acknowledged and addressed to ensure meaningful clinical advancement
