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However, Aja and colleagues found no evidence for this hypothesis since in this model CPT-1 should inhibit feeding by increasing cytosolic fatty-acyl CoA levels while they actually showed the initial response of mice to a CPT-1 inhibitor was an increase in appetite [75]

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Five of the 11 ACADs are involved in mitochondrial -oxidation: very long-chain acyl-CoA dehydrogenase (VLCAD), long-chain acyl-CoA (LCAD), medium-chain acyl-CoA dehydrogenases (MCAD), short-chain acyl-CoA dehydrogenase (SCAD), and ACAD9 (316)