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Nevertheless, there are no data on whether the effects of the immune-inflammatory processes during acute infection (as indicated by lowered SpO2 and increased body temperature) on the neuropsychiatric symptoms are mediated by activated immune and O&NS (IO&NS) pathways

Additional theories on the Multiple-Hit Hypothesis of MASLD detail the role of lipid and sugar metabolism, gut barrier dysfunction, and systemic inflammation as common factors in metabolic syndrome [233] MASLD/MASH, caused by non-alcohol substances, such as fructose/sucrose and Western-style high-fat diets (HFDs) (containing high ratios of pro-inflammatory omega-6 fatty acids to anti-inflammatory omega-3 fatty acids), is a hepatic manifestation of metabolic syndrome [234]

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