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Romanazzo et al
2017), bone and cartilage protection (Lim et al
Taken together, these results indicated that the C-terminal domain of AcvB mediates the interaction with LpiA and that LpiA promotes AcvB membrane association independently from the LpiA catalytic activity and the cellular Lys-PG level
In fact, transient overexpression of EAAT3 in hippocampal HT22 cells led to increased intracellular GSH levels in the presence of high glutamate concentrations, offering protection against oxidative glutamate toxicity [44, 45]
