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Conversely, the dynein mediates retrograde transport of various cargoes, and ALS-related mutations lead to transport abnormalities, axonal swelling and ultimately motor neuron degeneration ( The abnormal accumulation of proteins in motor neurons is a hallmark pathological feature of ALS, mainly including transactive response DNA-binding protein 43 (TDP-43), fused in sarcoma (FUS), SOD1 and sequestosome 1 (SQSTM1)

This can be explained by the difference in reaction rate between HOCl with methionine (which is relatively high) and the rate between HOCl with albumin (which is relatively low)

"Cirrhosis" in its previous name has been replaced by a more appropriate word, "cholangitis" in the current name because patients with this disease are increasingly diagnosed in the early stages of the disease before developing cirrhosis [123]