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Description
Given their role in disease progression, modulating macrophage phenotypic transformation and simultaneously inhibiting CCR2 and CCR5 represent promising therapeutic strategies for ALD

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H2S is another DNA-damaging mutagen, but it exerts an anti-inflammatory effect by facilitating the polarization of M2 macrophages, i.e., it has the advantage of both NO and CO, which can diminish the negative effects of PTT while improving the therapeutic effect and eliminating the biofilm

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