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Reversely, autophagy promotes NOXA degradation through p62
However, there is some concern that patients with massive acetaminophen overdoses will need more than the standard NAC treatment [81-83]
Mutants of the candidate residues were subsequently compared with wild-type ScGrx7 in steady-state kinetic measurements using HEDS and GSSCys as a non-glutathione and glutathione disulfide substrate, respectively
