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We also discuss the potential targets of ferroptosis in the treatment of cardiovascular disease and describe the current limitations and future directions of these novel treatment targets

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A polyclonal peptide Ab against the major immunogenic region in P/Q-type VGCC (the extracellular domain-III S5-6 loop) impaired the P/Q-type VGCC and caused a decrease in Ca 2+ currents, impairing synaptic transmission between PF and PC [44]