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MOG-targeted microparticle platforms have also been employed for deletion of pathogenic T cells to restore tolerance, an approach that contrasts with nanoscale systems that rely on intracellular uptake and cellular reprogramming (181)

18 TGF- functions through its interaction with TGF- receptors, which phosphorylate SMAD2/3, allowing these proteins to form complexes with SMAD4 that translocate to the nucleus and regulate gene expression

The key consideration is that Dihexa's effects persist beyond compound clearanceshifting protocols mid-study doesn't create an immediate reset but rather modulates ongoing neuroplastic processes already in motion

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