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doi: 10.1080/09168451.2015.1135042

NT disrupts the hydrogen bonds of proteins, impairing protein function [156, 157]

We describe the inward-facing structure as substrate-free rather than apo, as we observe weak, non-proteinaceous density in the cleft between the two domains (Supplementary Fig

Finally, interventions targeting the promotion of mitochondrial biogenesis and the restoration of mitochondrial function are much awaited
