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To deepen assess the impact of gpoA deletion on bacterial pathogenesis, we examined the intracellular survival of the gpoA mutant in three different host cell types: human pneumocytes (A549
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however, the ATP pool that is depleted in H 2 O 2 -induced necrosis is not affected in ferroptosis ( ATP5G3 , TPR repeat protein 35, citrate synthase and acyl-CoA synthetase family member 2, which are different from those involved in staurosporine-induced apoptosis ( To discuss how ferroptosis is involved in the pathogenesis of autoimmune diseases, some prototypic autoimmune diseases were chosen based upon the accessible messages from published work