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While receptor depletion from the plasma membrane occurs due to the -arresitin binding-induced endocytosis [26], it is reasonable to assume that the receptors are still synthesized and replenished with a rate r s , which leads to the following evolution equation for the receptor density on the cell surface: Dephosphorylation and resensitization of receptor In the longer time scale, it was suggested that typically only after the receptor internalization, protein phosphotases (PPs) are at work in an acidic endosomal compartment to dephosphorylate, resensitize and restore the receptor back to the cell surface [48, 50, 56, 57]

Patients with MCS exhibit elevated oxidative status and inflammatory markers compared to their healthy counterparts.[ref] MCS patients also have lower glutathione levels.[ref] Glutathione is a naturally produced antioxidant and one of the main ways that our cells fight oxidative stress

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These studies can be useful for understanding how receptor activation translates into signaling persistence and downstream program selection in controlled model systems