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Taurine emerges as a key protective factor against the activation of autophagy in the cerebrum of arsenic oxidoarsenous trioxide (As2O3)-intoxicated mice (103), given that the self-degradation process of autophagy is the main cause of arsenic-induced neurotoxicity (104,105)

In addition to the electrophoresis in the preexisting pores, there can generally be a significant (10 to 100 fold enhancement) contribution to transport enhancement arising from new pore induction (electroporation) (15), changes in pore sizes and flux in shunt pathways (16) ( e.g