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CarrASpencerEDasAMeijerNLaurenCMacPhersonSet alPatients undergoing myeloablative chemotherapy and hematopoietic stem cell transplantation exhibit depleted vitamin C status in association with febrile neutropenia

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By the physicians of Amanecia Health | Updated April 27, 2026 Last reviewed June 18, 2026 by Ann Czarnik, M.D., FACEP

Declared that transmembrane member 16A (TMEM16A), a component of hepatocyte Ca 2+ -activated chloride channel, exacerbates hepatic I/R injury through the promotion of GPX4-dependent ferroptosis, and interrupting the TMEM16A-GPX4 interaction or inhibiting TMEM16A in liver cells may represent promising therapeutic strategies for the treatment of hepatic I/R injury [416]