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Nevertheless, experimental data suggest that the altered intracellular redox status associated with hyperhomo-cysteinemia-induced oxidant stress is the major molecular mechanism responsible for the atherothrombogenicity of moderately elevated circulating homocysteine

After about 21% initial loss, participants who continued lost another 5.5%

As a result, SMARCA2 binds to the enhancers of the thyroid differentiation transcription factors (TFs) PAX8 and FOXE1, enhancing their expression by increasing chromatin accessibility

Specifically, IKE exerts anti-cancer activity by inducing lipid peroxidation and upregulating ferroptosis genes, including SLC7A11 (73)