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Necrosis and apoptosis after retinal ischemia: involvement of NMDA-mediated excitotoxicity and p53
doi: 10.1021/acs.jafc.9b08309 34
After combined use, it further promotes the decrease of SLC7A11 expression to promote ferroptosis and enhance the drug effect [28]
This factor (STAT) becomes activated via the upregulation of by Janus kinases (JAK), or the Src family kinases, thus allowing STAT to dimerize and translocate while binding to the promoters of target genes [161, 162]
