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This is why the reported effects are not just pigmentation

[14] Both formulations target patients inadequately controlled on metformin or other oral agents, with semaglutide functioning as a glucagon-like peptide-1 (GLP-1) receptor agonist to enhance insulin secretion, suppress glucagon release, and slow gastric emptying in a glucose-dependent manner

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While intriguing, these benefits lack robust clinical backing, and their consistency is uncertain
