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Abstract Although most patients with relapsed/refractory B-cell acute lymphoblastic leukemia (B-ALL) receiving CD19-targeted chimeric antigen receptor (CAR) T cell therapy achieve remission, loss of CAR T cell functionality and subsequent relapse remains an unmet therapeutic need
Results: Our findings indicate that 1) Prls capacity to antagonize cortisol-stimulated cftr expression depends on extracellular osmotic conditions
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