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Improving the integrity of this barrier is critical to minimizing inflammatory responses
Thus, the complex of the drug and MAbs has a targeted action, the ability to penetrate the BBB, and a therapeutic effect on the necessary elements of the brain: damaged neurons and tumor cells [194]

Recent research has demonstrated that plant-based dietary compounds can modulate NRF2 expression via epigenetic modifications

Therefore, targeting the HOTAIR/UPF1/ACSL4 axis is an effective strategy to inhibit ferroptosis and reduce neuronal death in ICH (Jin et al., 2021)
