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Our results provide a possible mechanism in which upregulation of phosphorylated p53 and p16 maintain the cell cycle arrest of keloid fibroblasts, and FOXO4-DRI selectively induces apoptosis of keloid senescent fibroblasts by prompting p53-pS15 nuclear exclusion
Enzymatic Degradation Although drugs administered to the nasal cavity can avoid gastrointestinal degradation and the hepatic first-pass effect, the nasal cavity contains hydrolytic enzymes as part of its defense mechanism against xenobiotics, leading to enzymatic drug degradation
Hyperactive intracellular calcium dynamics in B lymphoblasts from patients with bipolar I disorder
Other toxic molecules are then formed, leading to tissue, cellular, and DNA damage [13, 15]
