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Additionally, the integration of computational modeling and AI-driven design tools could further optimize the binding affinity, specificity, and degradation efficiency of future constructs, enabling personalized therapeutic approaches based on individual tumor profiles

Mitochondrial stress markers VDAC1 and NADPH oxidase 4 were downregulated

Treatment with exogenous GSH during acute infections or illnesses can amplify immune response by restoring optimum redox conditions for adequate immune cell signal transduction (Diotallevi et al., 2017)

GBM cells increase conversion of tryptophan to kynurenine via upregulation of indoleamine 2,3-dioxygenase (IDO1/IDO2) and tryptophan 2,3-dioxygenase (TDO2), depleting local levels of tryptophan and impairing T-cell functioning (107, 108)