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L-theanine, in contrast, promotes relaxation without sedation by increasing alpha brain waves and modulating GABA, serotonin, and dopamine activity

During sustained ERS, PERK phosphorylates eukaryotic initiation factor 2 (eIF2), leading to activating transcription factor 4 (ATF4)-mediated upregulation of the pro-apoptotic transcription factor CHOP (90, 91)

Additionally, using the same animal model, AEA levels are increased while FAAH activity is reduced in the striatum, supporting a boost of the ECS, and probably reflecting a compensatory mechanism to counteract chronic dopamine depletion (Gubellini et al., 2002)

doi: 10.1007/s11095-011-0551-1