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The GSTO1 C1-27 and GSTO1 C1-31 crystals were cryoprotected with 30% PEG and 100 mM MES (pH 6.5) before data collection

doi: 10.1016/j.colsurfb.2026.115538 99 GuerriniL.Alvarez-PueblaR

According to the current paradigm, glutamate-driven excitotoxicity, activation of calcium-dependent proteases, over activation of calcium-regulated kinases and phosphatases, and mitochondrial calcium overload resulting in mPTP opening (53, 54) are some of the mechanisms by which generalized calcium overload in neurons causes neuronal death (54, 55)

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