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Therefore, sphingolipid-autophagy crosstalk constitutes a pivotal regulatory nexus governing Treg stability, whose diabetic disruption propagates T1DM immunopathology by crippling immunosuppressive circuits, while pharmacologically restoring this axis emerges as a strategic therapeutic paradigm for re-establishing immune tolerance (Figure 5)

Curcumin improves insulin receptor signaling, preserves beta-cell function, regulates adipokine production by increasing adiponectin and decreasing leptin and resistin, and alters miRNA expression, including miR-21, miR-146a, and miR-155, to mitigate neuroinflammation and promote neuronal repair

Strickler, J.E