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The depletion of GSH may also allow viruses to replicate, and therefore is a major factor in the pathology of COVID-19 [36]

Importantly, there is functional crosstalk between NO signaling and TRPV1 activation: NO can modulate TRPV1 activity indirectly through protein kinase pathways, while TRPV1-driven calcium influx influences downstream NO production [91,92]

Int J Med Sci 15:1015

Exclusion criteria included manuscripts in languages other than English and those with incomplete documentation
