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doi: 10.1210/clinem/dgab456 49 MissoG

These findings suggest a close relationship between METH-induced perturbations of energy metabolism and dopaminergic neurotoxicity, and that potential therapeutic strategies might bypass bioenergetic defects if defective mitochondrial energy metabolism has a role in neurotoxicity
Traditional supplementation practices, often characterized by heterogeneous results and variable clinical utility, are increasingly being replaced by evidence-informed frameworks integrating molecular insights, nutrigenomics, and translational trial design

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