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Early studies demonstrated that complement-mediated damage to the postsynaptic NMJ results in reduction of the postsynaptic junctional folds, elimination of AChR from the membrane, and an increase in synaptic distance (59, 60)
doi: 10.1007/s00403-024-02988-0
Further studies indicated its possible role in the cleavage of glutathione in periplasmic space to provide cysteine and glycine in Gram-negative bacteria, similar to mammalian GGTs
