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Recently, Ebrahimi, Rostam-Abadia, and Rezaei [131] reviewed and highlighted a growing body of research detailing NK cell dysfunction in children with ASD as well as their parents

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Therefore, this criterion was deemed conclusive enough to discount the reduced model as a viable model for describing the OCR data, but insufficient to prompt selection of either the triad or comproportionation model

In parallel, inactivity can enhance non-mitochondrial reactive oxygen species production through NADPH oxidases, disrupt calcium homeostasis, and impair mitochondrial quality-control mechanisms such as mitophagy