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ANG was identified as a molecule involved in angiogenesis because itoccurred as a result of a hit here to VEGF, a prototypical course of VEGF that ultimately emerged as a molecule with a prior neuroprotective classification

This therapy obviously needs to be more investigated to avoid side effects in normal tissues but the combination of specific senescence markers to cytotoxic drugs is a potential promising therapy

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The FDA does not have to follow PCAC recommendations, but it almost always does and when it doesnt, the divergence becomes its own news