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These methylation alterations are often duration- and sex-dependent and relate to accelerated biological aging, disrupted circadian regulation, immune dysfunction, and potentially increased BC risk

Glia 62 : 272283

The age at onset as well as the rate of progression of the dying-back axonopathy can vary dramatically

M(6)A Demethylase ALKBH5 Maintains Tumorigenicity of Glioblastoma Stem-Like Cells by Sustaining FOXM1 Expression and Cell Proliferation Program
