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PMA is known to trigger a NOX2-dependent oxidative burst via the activation of protein kinase C (PKC) and mitogen-activated protein kinases (MAPKs), both of which are critical for NET formation 35

Thus, it is hypothesized that CBS can heighten the susceptibility of STAD cells to ferroptosis through GSH depletion

Recently, the purpurin analog R162 (an inhibitor of GLUD1) also showed promising results with respect to attenuating the proliferation of breast, NSCLC, and glioma cells in vitro and in patient-derived xenograft mouse models 23

Injectable glutathione offers the highest bioavailability but comes with increased risks and should only be administered by qualified healthcare professionals in appropriate medical settings