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Description
Thus, given the particularities of the observed BPC 157 therapy effect [1,2,3,4,5,6,7,8,9,10,11,15,27,28,29,30,33,109,124,125], this final point, the equation between the exogenous effects and endogenous presence to verify endogenous presence and significance, can be a suggestive argument for the possible BPC 157/neurotransmitter relation
SAM, a key methyl donor, is derived from folate/one-carbon metabolism and the methionine cycle, with its levels regulated by mitochondrial metabolism, thereby controlling the supply of methylated substrates [250]
Located at 4001 E
R.et al (2011b)
