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Notably, SLC27A5 negatively correlates with GSR, and genetic or pharmacological inhibition of GSR strengthens the efficacy of sorafenib through GSH depletion and the accumulation of lipid peroxide products in SLC27A5-knockout and sorafenib-resistant HCC cells
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Another strategy involves switching to selective COX-2 inhibitors like celecoxib, which spare the constitutively expressed gastrointestinal enzymes while providing targeted anti-inflammatory effects
