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Autism-associated neuroligin-3 mutations commonly disrupt tonic endocannabinoid signaling

This mechanism is particularly critical, as PGE2 not only directly participates in inflammatory responses but also sensitizes sensory neurons via TRPV1 channel activation, thereby inducing the release of TNF-, interleukin-6, and vascular endothelial growth factor, which exacerbate vasodilation and non-neurogenic inflammation (Sulzberger et al., 2016)

For example, it reacts with nitric oxide with a formation of peroxynitrite (ONOO-), a strong oxidant, which leads to formation of reactive intermediates due to spontaneous decomposition [9-10]

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