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doi: 10.5607/en.2013.22.1.11
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Research has characterised whether the two distinct angiogenic mechanisms , VEGF/NO-dependent endothelial activation and PI3K/Akt-dependent endothelial migration , produce additive or synergistic new vessel formation in ischaemia and wound healing models, contributing to understanding of the mechanistic complementarity of the two angiogenic pathways
A 2015 paper on GHK as a natural modulator of skin-regeneration pathways similarly maps multiple cellular pathways, again in models rather than large human trials (PMC4508379)
