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Description
Conventional treatments fail to regenerate damaged tissues or restore immune tolerance
[19] Additionally, excess mitochondrial ROS activates caspase-3 and the mitochondrial apoptotic cascade by increasing thioredoxin-interacting protein expression, contributing to beta cell loss in young New Zealand obese mice
sits on the science advisory boards of the Deutsches Konsortium fr Translationale Krebsforschung (DKTK) and the Nationales Centrum fr Tumorerkrankungen (NCT)
This approach takes advantage of differences between healthy and malignant cells, such as the absence of a drug target
