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The mechanism for the formation of may proceed via the initial attack of a water molecule at the selenium center in compound followed by a nucleophilic attack of the selenium-bound oxygen atom at the carbonyl carbon of one of the amide moieties, leading to the formation of an intermediate (Figure 8) [59]

Neuroprotective effect of curcumin against inhibition of mitochondrial complex I in vitro and in vivo.Implications for Parkinson's disease explained via in silico studies

Ubiquitin-mediated NFkappaB degradation pathway

The patient received two cycles of neoadjuvant chemotherapy combined with immunotherapy (tislelizumab + carboplatin + docetaxel)