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Description
These diseases are mainly represented by the congenital defects of urea cycle, classical organic acidurias, and the defects of mitochondrial fatty acids oxidation, with hyperammonemia being more severe and frequent in the first two groups mentioned
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If you have genetic raw data from 23andMe, AncestryDNA, or whole genome sequencing, the genotype report below checks your variants in immune, inflammatory, mitochondrial, and neurological pathways linked to ME/CFS susceptibility
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