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10.1158/1078-0432.CCR-18-3339 59 KobayashiH.ImanakaS.YoshimotoC.MatsubaraS.ShigetomiH
Consistent with these in vivo effects, we found that bradykinin-stimulated production of NO was enhanced by vector-mediated GPx1 overexpression in cultured endothelial cells at baseline and in the presence of exogenous homocysteine [60]
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In PD animal models, activating protective autophagy with mTOR inhibitors (such as rapamycin) while modulating microglial polarization and inhibiting inflammation with TREM2 agonists significantly improves motor function and neuropathological symptoms [315]
