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It is conceivable, but not yet demonstrated, that ferroptosis could be triggered during development or normal homeostatic tissue turnover by the accumulation of glutamate, iron, or PUFA-phospholipids, or by depletion of endogenous inhibitors of ferroptosis, such as GSH, NADPH, GPX4 or vitamin E
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Neuropathology of Alzheimers disease: a critical update
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