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Therefore, above we considered how TLR4 potentially can trigger LPS-induced inflammation caused by the disruption of intestinal integrity through the TIRAP/MyD88 signaling cascade, that leads to the early activation of NF-B (Figure 2, left part)

Macrophage-derived GSDMD plays an essential role in atherosclerosis and cross talk between macrophages via the mitochondria-STING-IRF3/NF-B axis

Induction of glutathione s-transferases and hepatocellular proliferating activities in the rat liver treated with tert-butylated hydroxyanisole, 1,2-bis(2-pyridyl)ethylene, and phenobarbital