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Abstract Background: The interplay between bacterial virulence factors and the host innate immune response in pneumococcal meningitis (PM) can result in uncontrolled neuroinflammation, which is known to induce apoptotic death of progenitor cells and post-mitotic neurons in the hippocampal dentate gyrus, resulting in cognitive impairment

It is studied in preclinical models to investigate selective lipolysis stimulation, adipocyte metabolism regulation, body composition effects, and metabolic parameter modulation

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