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Taken together, these new data demonstrate the complex interactions between NO, ROS signalling and the antioxidant system, although complementary investigations are needed to provide a global comprehension of this system

The designed peptide (referred to as VGB4) was able to bind to both VEGFR1 and VEGFR2 on the Human Umbilical Vein Endothelial Cells (HUVECs) surface and inhibited VEGF-A driven proliferation, migration and tube formation in HUVECs through suppression of ERK1/2 and AKT phosphorylation

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