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Conclusions In these studies, within the limitations of small sample sizes, no clinically relevant differences in cagrilintide pharmacokinetics were observed in participants with renal or hepatic impairment compared with those with normal function, suggesting that dose adjustment is not warranted for these populations

Thus, DN30 with dual effects on c-Met and HGF separately can inhibit the c-Met/HGF pathway [173]

When the thiol compound is deficient, DQ is spontaneously cyclized to leukodopa, which is further oxidized to dopachrome in concert with the reduction of DQ to DOPA [79]

doi: 10.7150/ijms.91940 Summary Keywords lung cancer, ferroptosis, cell death mechanisms, tumor microenvironment, therapeutic strategies, molecular pathways Citation Gao G and Zhang X (2025) Broadening horizons: research on ferroptosis in lung cancer and its potential therapeutic targets