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Introduction In 2003, erastin, a novel compound was identified, which showed a selective lethality against human foreskin cells (BJeLR cells) expressing engineered mutated RAS oncogene while using high-throughput screening to find out 23,550 compounds for their ability to eliminate engineered tumorigenic cells (Yang and Stockwell, 2008)

It further demonstrates the feasibility of developing actionable and highly precise functional interventions by integrating genomic and biochemical data

Shakt et al., 2024), and the vascular depression hypothesis ( The interaction mechanism between AS and depression is highly complex, involving multiple potential mechanisms, including inflammation (Paranthaman et al., 2012), immune responses (Mousa et al., 2022), autonomic nervous system dysfunction (Pizzi et al., 2010), and intestinal flora and metabolite dysfunction (Liao et al., 2023)

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