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Temozolomide Induces Senescence and Repression of DNA Repair Pathways in Glioblastoma Cells via Activation of ATRCHK1, p21, and NF-BMechanisms of Methylating Anticancer DrugInduced Senescence

Bd infection depletes host glutathione stores and increases ROS An in vitro cell infection model (25, 28) was used to further investigate the role of glutathione during chytridiomycosis

Wen-Jing Zeng: Writing review & editing, Investigation, Methodology, Conceptualization

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