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Although these observations suggest that LepR signalling might be a therapeutic target for treating obesity, how energy imbalance and obesity develop when endogenous leptin is functional is still puzzling
CrascLLauroMRPuglisiGPanicoA
doi: 10.4049/jimmunol.168.7.3451 61 MarcenaroEFerrantiBFalcoMMorettaLMorettaA
