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Description
Experimental GPX1 deficiency amplifies certain aspects of aging, namely endothelial dysfunction, vascular remodeling, and invasion of leukocytes in cardiovascular tissue
doi: 10.1021/cr0103773
Activation of NF-B also induces the upregulation of matrix metalloproteinase-9 (MMP-9) (94) and elevated mitochondrial MMP-9 levels cause damage to connexin-43, a mitochondrial gap junction protein, thereby increasing mitochondrial membrane permeability
Research findings: Cell culture studies show protected fibroblast viability under stress conditions
