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Since Pgp can be inhibited at transcriptional level by C/EBP- LIP [5] and at post-translational level by NO [14, 15], we set up a strategy that increases at the same time C/EBP- LIP and NO upon treatment with doxorubicin, in order to downregulate expression and activity of Pgp and overcome the resistance to the drug in TNBC

[DOI] [PMC free article] [PubMed] [Google Scholar] 400.Farkas T

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