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In a D-Asp oxidase knockout mouse model, treatment with D-Asp significantly alleviated phencyclidine-induced cortico-limbic thalamic dysfunction and reduced neuronal prepulse deficits induced by psychotropic drugs (amphetamine and MK-801)

Sensing and signaling of oxidative stress in chloroplasts by inactivation of the SAL1 phosphoadenosine phosphatase

(B) M pro activity for a WT monoglutathionylated M pro preparation (containing approximately 30% monoglutathionylated M pro and 4% diglutathionylated) and a C300S monoglutathionylated M pro preparation (containing approximately 18% monoglutathionylated M pro ) preincubated for 10 minutes without or with 20 mM DTT